I was born in Leon, Spain, where I did my BSc in Biology. I completed my PhD in Toledo, Spain, at the University of Castilla-La Mancha while I was working as an Exercise Physiology Lecturer’s Assistant.
I was fortunate to gain a postdoctoral fellowship to come to Australia to the Baker IDI Heart and Diabetes Institute in Melbourne to work under Prof Mark Febbraio’s supervision.
After three years working as a Research Officer I moved into lab management duties, tasks that I’ve been combining with both lab and animal work for the last three years. I have been appointed at the Garvan Institute as the Diabetes and Metabolism Division Manager.
Awards and Honours
2008 - Spanish National Award in Sports Medicine Research, 3rd place
2002-2004 - Scholarship for research (EFENVIR Study), Research Unit of the Hospital of Leon: Immunology. Bristol-Myers Squibb
2001-2002 - Scholarship for formation, Microscopy Unit of the University of Leon, University of Leon, Spain
2001 - BSc in Biology, Faculty of Biology and Environmental Sciences, University of León - Spain
Kraakman MJ, Kammoun HL, Allen TL, Deswaerte V, Henstridge DC, Estevez E, Matthews VB, Neill B, White DA, Murphy AJ, Peijs L, Yang C, Risis S, Bruce CR, Du XJ, Bobik A, Lee-Young RS, Kingwell BA, Vasanthakumar A, Shi W, Kallies A, Lancaster GI, Rose-John S, Febbraio MA. Blocking IL-6 trans-signaling prevents high-fat diet-induced adipose tissue macrophage recruitment but does not improve insulin resistance. Cell Metab. 2015 Mar 3;21(3):403-16. doi: 10.1016/j.cmet.2015.02.006.
Jordy AB, Kraakman MJ, Gardner T, Estevez E, Kammoun HL, Weir JM, Kiens B, Meikle PJ, Febbraio MA, Henstridge DC. Analysis of the liver lipidome reveals insights into the protective effect of exercise on high-fat diet-induced hepatosteatosis in mice. Am J Physiol Endocrinol Metab. 2015 May 1;308(9):E778-91. doi: 10.1152/ajpendo.00547.2014. Epub 2015 Feb 24
Henstridge DC, Estevez E, Allen TL, Heywood SE, Gardner T, Yang C, Mellett NA, Kingwell BA, Meikle PJ, Febbraio MA. Genetic manipulation of cardiac Hsp72 levels does not alter substrate metabolism but reveals insights into high-fat feeding-induced cardiac insulin resistance. Cell Stress Chaperones. 2015 May;20(3):461-72. doi: 10.1007/s12192-015-0571-6. Epub 2015 Jan 25.
Henstridge DC, Bruce CR, Drew BG, Tory K, Kolonics A, Estevez E, Chung J, Watson N, Gardner T, Lee-Young RS, Connor T, Watt MJ, Carpenter K, Hargreaves M, McGee SL, Hevener AL, Febbraio MA. Activating HSP72 in rodent skeletal muscle increases mitochondrial number and oxidative capacity and decreases insulin resistance. Diabetes. 2014 Jun;63(6):1881-94. doi: 10.2337/db13-0967. Epub 2014 Jan 15
Lindegaard B, Matthews VB, Brandt C, Hojman P, Allen TL, Estevez E, Watt MJ, Bruce CR, Mortensen OH, Syberg S, Rudnicka C, Abildgaard J, Pilegaard H, Hidalgo J, Ditlevsen S, Alsted TJ, Madsen AN, Pedersen BK, Febbraio MA. Interleukin-18 activates skeletal muscle AMPK and reduces weight gain and insulin resistance in mice. Diabetes. 2013 Sep;62(9):3064-74. doi: 10.2337/db12-1095. Epub 2013 May 13.
Dr Emma EstevezEmail: Click here to Email