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Insulin action under arrestin


Insulin signaling is key to the etiology of metabolic syndrome. Recent work (Luan et al., 2009) uncovers a role for beta-arrestin, previously known to control GPCR desensitization, in insulin signaling. In mouse models, beta-arrestin-2 controls whole-body insulin action by regulating assembly of a complex containing insulin receptor, c-Src, and Akt.

Type Journal
ISBN 1932-7420 (Electronic)
Authors Stockli, J.; James, D. E.;
Garvan Authors
Publisher Name CELL METAB
Published Date 2009-01-01 00:00:00
Published Volume 9
Published Issue 3
Published Pages 213-4
Status Published In-print
OpenAccess Link Stockli CM.pdf