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Insulin action under arrestin

Abstract

Insulin signaling is key to the etiology of metabolic syndrome. Recent work (Luan et al., 2009) uncovers a role for beta-arrestin, previously known to control GPCR desensitization, in insulin signaling. In mouse models, beta-arrestin-2 controls whole-body insulin action by regulating assembly of a complex containing insulin receptor, c-Src, and Akt.

Type Journal
ISBN 1932-7420 (Electronic)
Authors Stockli, J.; James, D. E.;
Responsible Garvan Author
Publisher Name Cell Metabolism
Published Date 2009-01-01 00:00:00
Published Volume 9
Published Issue 3
Published Pages 213-4
Status Published in-print
URL link to publisher's version http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&dopt=Citation&list_uids=19254565
OpenAccess link to author's accepted manuscript version https://publications.gimr.garvan.org.au/open-access/10231